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ImmuneCited

Proceso de investigación

333 figuras de investigación revisada por expertos

Todos Pelargonium sidoides Glutathione N-Acetylcysteine Astragalus Conjugated Linoleic Acid Curcumin Arginine Astaxanthin Cordyceps Omega-3 Fatty Acids (EPA/DHA) Zinc Quercetin Elderberry Resveratrol Vitamin D Andrographis Iron
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Figure 3
Figure 3 Chart

Viral titer decay curves on stainless steel and other metal surfaces demonstrate that coronavirus 229E can remain infectious for several days under ambient conditions.

Human Coronavirus 229E Remains Infectious on Common Touch Surface Materials.

Figure 4
Figure 4 Chart

Comparison of coronavirus inactivation rates across polymer-based surfaces including PVC, silicone rubber, and Teflon shows material-dependent variation in viral survival.

Human Coronavirus 229E Remains Infectious on Common Touch Surface Materials.

Figure 5
Figure 5 Chart

Temperature and humidity effects on coronavirus 229E survival on surfaces are displayed, with higher temperatures generally associated with faster viral inactivation.

Human Coronavirus 229E Remains Infectious on Common Touch Surface Materials.

Figure 6
Figure 6 Photograph

Scanning electron microscopy or surface characterization images of the tested materials are presented alongside their corresponding viral persistence data.

Human Coronavirus 229E Remains Infectious on Common Touch Surface Materials.

Figure 7
Figure 7 Chart

Recovery efficiency controls for the surface sampling methodology are shown, validating the quantitative viral titer measurements across different material types.

Human Coronavirus 229E Remains Infectious on Common Touch Surface Materials.

Figure 8
Figure 8 Chart

Summary comparison of coronavirus 229E persistence across all tested surface materials, ranked by duration of detectable infectivity.

Human Coronavirus 229E Remains Infectious on Common Touch Surface Materials.

Figure 1. Chemical structure of quercetin.
Figure 5 Diagram

Chemical structure of quercetin (3,3',4',5,7-pentahydroxyflavone) is displayed, showing the hydroxyl groups on the flavonoid backbone that contribute to its antioxidant and anti-allergic properties.

Quercetin and Its Anti-Allergic Immune Response.

Figure 3
Figure 3 Diagram

Pathophysiological mechanisms shared between osteoporosis and sarcopenia are diagrammed, including hormonal, nutritional, and mechanical loading factors.

Osteosarcopenia: epidemiology, diagnosis, and treatment-facts and numbers.

Figure 4
Figure 4 Flowchart

Treatment algorithms for osteosarcopenia incorporating exercise, nutritional supplementation, and pharmacological interventions are outlined.

Osteosarcopenia: epidemiology, diagnosis, and treatment-facts and numbers.

Figure 5
Figure 5 Chart

Outcome data from intervention studies targeting both bone and muscle health in osteosarcopenic patients are compared across different therapeutic approaches.

Osteosarcopenia: epidemiology, diagnosis, and treatment-facts and numbers.

Figure 2. Phases and specific events of the wound healing process.
Figure 6 Diagram

Sequential phases of the wound healing process - hemostasis, inflammation, proliferation, and remodeling - and their specific cellular events are illustrated.

Nutrition and Wound Healing: An Overview Focusing on the Beneficial Effects of …

Figure 3. Structure and effects of curcumin on wound healing.
Figure 7 Diagram

Chemical structure of curcumin alongside its documented effects on wound healing, including anti-inflammatory, antioxidant, and antimicrobial activities, are presented.

Nutrition and Wound Healing: An Overview Focusing on the Beneficial Effects of …

Figure 1. The proposed mechanism by which astaxanthin inhibits oxidative stress-induced mitochondrial dysfunction, and development and progression of diseases.
Figure 5 Diagram

Proposed comprehensive mechanism by which astaxanthin inhibits oxidative stress-induced mitochondrial dysfunction, preventing downstream apoptotic and inflammatory signaling cascades.

Inhibitory Effect of Astaxanthin on Oxidative Stress-Induced Mitochondrial Dysfunction-A Mini-Review.

Figure 1. Some of the molecular bases of resveratrol anti-inflammatory effects. Inflammation induces the activation of several cell signaling pathways. The exact mechanism of RSV-mediated protection is not yet understood, but it was described that RSV int
Figure 5 Diagram

Molecular bases of resveratrol's anti-inflammatory effects are comprehensively illustrated, showing how inflammation activates multiple signaling pathways that are concurrently modulated by resveratrol through NF-kB, MAPK, and SIRT1 mechanisms.

Anti-Inflammatory Effects of Resveratrol: Mechanistic Insights.

Figure 1
Figure 1 Diagram

Multiple convergent pathways leading to mitochondrial dysfunction in neuroimmune and neuropsychiatric disorders are mapped, including oxidative stress, calcium dysregulation, and inflammatory cytokine signaling.

The many roads to mitochondrial dysfunction in neuroimmune and neuropsychiatric disorders.

Figure 5
Figure 5 Diagram

Gut microbiome composition alterations in RA patients and how dietary prebiotics and probiotics may influence disease progression.

Dietary Habits and Nutrition in Rheumatoid Arthritis: Can Diet Influence Disease Development …

Figure 6
Figure 6 Diagram

Vitamin D metabolism and its immunomodulatory roles relevant to rheumatoid arthritis pathogenesis are diagrammed.

Dietary Habits and Nutrition in Rheumatoid Arthritis: Can Diet Influence Disease Development …

Figure 7
Figure 7 Diagram

Nutrients and their food sources involved in RA development and progression are mapped, with asterisks indicating nutrients with less well-defined evidence for their role in the disease.

Dietary Habits and Nutrition in Rheumatoid Arthritis: Can Diet Influence Disease Development …

Figure 2. Nutrients and their food sources involved in the development and progression of Rheumatoid Arthritis. * Nutrients with less defined evidence. TMAO: trimethylamine-N-oxide.
Figure 8 Diagram

Polyphenol-rich foods and their documented anti-inflammatory mechanisms relevant to rheumatoid arthritis management are categorized.

Dietary Habits and Nutrition in Rheumatoid Arthritis: Can Diet Influence Disease Development …

Figure 1. Chemical structure of (A) curcumin, (B) bis-demethoxycurcumin, (C) demethoxycurcumin. Structures were drawn using ChemSpider, an online free chemical structure database.
Figure 6 Diagram

Chemical structures of curcumin, bis-demethoxycurcumin, and demethoxycurcumin - the three main curcuminoids - are displayed, highlighting the structural differences that influence their biological activity.

The Role of Curcumin in Cancer Treatment.

Figure 2. Summarized effects of curcumin on cancer cells. Key: CDK2, cyclin-dependent kinase 2; MMP-2, matrix metallopeptidase 2; MMP-9, matrix metallopeptidase 9; VEGF, vascular endothelial growth factor; NF-kb, nuclear factor kappa-light-chain-enhancer
Figure 7 Diagram

Summarized effects of curcumin on cancer cells are comprehensively mapped, showing modulation of CDK2, MMP-2, MMP-9, and multiple other molecular targets involved in tumor growth and metastasis.

The Role of Curcumin in Cancer Treatment.

Figure 1
Figure 1 Forest Plot

Meta-analysis results indicate that vitamin D deficiency is significantly more prevalent among patients with autoimmune thyroid disease compared to healthy controls. Lower serum 25-hydroxyvitamin D levels are associated with increased odds of both Hashimoto's thyroiditis and Graves' disease.

Meta-analysis of the association between vitamin D and autoimmune thyroid disease.

Fig. 1 Histopathological alterations in ovarian sections of the control and the experimental groups. a control; b PCOS; c solvent; d resveratrol; e metformin and f metformin+resveratrol group. g-l higher magnification of the follicles in these groups, res
Figure 5 Micrograph

Histopathological analysis of ovarian sections across control and experimental groups shows characteristic PCOS features including multiple cystic follicles and thickened theca cell layers. Treatment groups demonstrate partial normalization of ovarian morphology.

Effect of resveratrol and metformin on ovarian reserve and ultrastructure in PCOS: …

Fig. 2 A Representative SIRT1 immunreactivity in control, PCOS, resveratrol, metformin and metformin+resveratrol groups. a; control, b; PCOS, c; solvent, d; resveratrol, e; metformin and f; metformin+resveratrol groups. B HSCORE of SIRT1 immunostaining in
Figure 6 Micrograph

SIRT1 immunoreactivity in ovarian tissue varies across experimental groups, with reduced expression in PCOS rats and partial restoration following resveratrol and metformin treatment. SIRT1 is implicated in cellular stress responses and metabolic regulation.

Effect of resveratrol and metformin on ovarian reserve and ultrastructure in PCOS: …

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