Descripción

Intracellular reactive oxygen species (ROS) levels in IPEC-J2 cells, showing that astaxanthin co-treatment reduced the AFB1-induced elevation in ROS, supporting its antioxidant protective role.

Figure 12

Chart

Source Paper

Astaxanthin Alleviates Aflatoxin B1-Induced Oxidative Stress and Apoptosis in IPEC-J2 Cells via the Nrf2 Signaling Pathway.

Toxins (2023)

PMID: 36977123

DOI: 10.3390/toxins15030232

Cite This Figure

![Figure 12: Intracellular reactive oxygen species (ROS) levels in IPEC-J2 cells, showing that astaxanthin co-treatment reduced the AFB1-induced elevation in ROS, supporting its antioxidant protective role.]()

> Source: Yue Tian et al. "Astaxanthin Alleviates Aflatoxin B1-Induced Oxidative Stress and Apoptosis in IP." *Toxins*, 2023. PMID: [36977123](https://pubmed.ncbi.nlm.nih.gov/36977123/)

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  <img src="" alt="Intracellular reactive oxygen species (ROS) levels in IPEC-J2 cells, showing that astaxanthin co-treatment reduced the AFB1-induced elevation in ROS, supporting its antioxidant protective role." />
  <figcaption>Figure 12. Intracellular reactive oxygen species (ROS) levels in IPEC-J2 cells, showing that astaxanthin co-treatment reduced the AFB1-induced elevation in ROS, supporting its antioxidant protective role.<br>  Source: Yue Tian et al. "Astaxanthin Alleviates Aflatoxin B1-Induced Oxidative Stress and Apoptosis in IP." <em>Toxins</em>, 2023. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/36977123/">36977123</a></figcaption>
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