Figure 2

![Figure 2: Molecular pathway analysis from the liraglutide wound healing study identifying Myo1c/Dock5 as key mediators. Protein expression or activation levels are quantified to establish the mechanistic link between GLP-1 receptor agonism and tissue repair.]()

> Source: Qian Zhang et al. "Liraglutide Promotes Diabetic Wound Healing via Myo1c/Dock5.." *Advanced science (Weinheim, Baden-Wurttemberg, Germany)*, 2024. PMID: [39159301](https://pubmed.ncbi.nlm.nih.gov/39159301/)

<figure>
  <img src="" alt="Molecular pathway analysis from the liraglutide wound healing study identifying Myo1c/Dock5 as key mediators. Protein expression or activation levels are quantified to establish the mechanistic link between GLP-1 receptor agonism and tissue repair." />
  <figcaption>Figure 2. Molecular pathway analysis from the liraglutide wound healing study identifying Myo1c/Dock5 as key mediators. Protein expression or activation levels are quantified to establish the mechanistic link between GLP-1 receptor agonism and tissue repair.<br>  Source: Qian Zhang et al. "Liraglutide Promotes Diabetic Wound Healing via Myo1c/Dock5.." <em>Advanced science (Weinheim, Baden-Wurttemberg, Germany)</em>, 2024. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/39159301/">39159301</a></figcaption>
</figure>