Arginine methylation in the epithelial-to-mesenchymal transition.
Study Design
- Study Type
- Review
- Population
- None
- Intervention
- Arginine methylation in the epithelial-to-mesenchymal transition. None
- Comparator
- None
- Primary Outcome
- None
- Effect Direction
- Mixed
- Risk of Bias
- Unclear
Abstract
Epithelial cells acquire mesenchymal characteristics during embryonic development, wound healing, fibrosis, and in cancer in a processed termed epithelial-to-mesenchymal transition (EMT). Regulatory networks of EMT are controlled by post-transcriptional, translational, and post-translational mechanisms, in which arginine methylation is critically involved. Here, we review arginine methylation-dependent mechanisms that regulate EMT in the aspects of signaling, transcriptional, and splicing regulation.
TL;DR
Arginine methylation‐dependent mechanisms that regulate EMT in the aspects of signaling, transcriptional, and splicing regulation are reviewed.
Used In Evidence Reviews
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