描述
Mitochondrial dysfunction in retinal pigment epithelial cells is illustrated as a key driver of dry age-related macular degeneration progression, linking Dicer deficiency to Alu RNA-mediated damage.
Figure 1
DiagramSource Paper
Nutraceuticals/Drugs Promoting Mitophagy and Mitochondrial Biogenesis May Combat the Mitochondrial Dysfunction Driving Progression of Dry Age-Related Macular Degeneration.Cite This Figure
![Figure 1: Mitochondrial dysfunction in retinal pigment epithelial cells is illustrated as a key driver of dry age-related macular degeneration progression, linking Dicer deficiency to Alu RNA-mediated damage.]() > Source: Lidianys María Lewis Luján et al. "Nutraceuticals/Drugs Promoting Mitophagy and Mitochondrial Biogenesis May Combat." *Nutrients*, 2022. PMID: [35565950](https://pubmed.ncbi.nlm.nih.gov/35565950/)
<figure> <img src="" alt="Mitochondrial dysfunction in retinal pigment epithelial cells is illustrated as a key driver of dry age-related macular degeneration progression, linking Dicer deficiency to Alu RNA-mediated damage." /> <figcaption>Figure 1. Mitochondrial dysfunction in retinal pigment epithelial cells is illustrated as a key driver of dry age-related macular degeneration progression, linking Dicer deficiency to Alu RNA-mediated damage.<br> Source: Lidianys María Lewis Luján et al. "Nutraceuticals/Drugs Promoting Mitophagy and Mitochondrial Biogenesis May Combat." <em>Nutrients</em>, 2022. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/35565950/">35565950</a></figcaption> </figure>