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Summary model of the proposed mechanism by which Bifidobacterium lactis BB-12 attenuates macrophage aging and promotes M2 polarization. The diagram integrates oxidative stress reduction, inflammatory modulation, and polarization shifting pathways.
Figure 9
DiagramSource Paper
Bifidobacterium lactis BB-12 Attenuates Macrophage Aging Induced by D-Galactose and Promotes M2 Macrophage Polarization.Cite This Figure
![Figure 9: Summary model of the proposed mechanism by which Bifidobacterium lactis BB-12 attenuates macrophage aging and promotes M2 polarization. The diagram integrates oxidative stress reduction, inflammatory modulation, and polarization shifting pathways.]() > Source: Da-Yong Zhang et al. "Bifidobacterium lactis BB-12 Attenuates Macrophage Aging Induced by D-Galactose ." *Journal of immunology research*, 2019. PMID: [31930149](https://pubmed.ncbi.nlm.nih.gov/31930149/)
<figure> <img src="" alt="Summary model of the proposed mechanism by which Bifidobacterium lactis BB-12 attenuates macrophage aging and promotes M2 polarization. The diagram integrates oxidative stress reduction, inflammatory modulation, and polarization shifting pathways." /> <figcaption>Figure 9. Summary model of the proposed mechanism by which Bifidobacterium lactis BB-12 attenuates macrophage aging and promotes M2 polarization. The diagram integrates oxidative stress reduction, inflammatory modulation, and polarization shifting pathways.<br> Source: Da-Yong Zhang et al. "Bifidobacterium lactis BB-12 Attenuates Macrophage Aging Induced by D-Galactose ." <em>Journal of immunology research</em>, 2019. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/31930149/">31930149</a></figcaption> </figure>