Descrição

Proposed pathogenic model linking anti-selenoprotein P autoantibodies to chronic fatigue syndrome symptoms through disrupted selenium transport, impaired deiodinase activity, and functional thyroid hormone resistance.

Figure 6

Diagram

Source Paper

Autoantibodies to selenoprotein P in chronic fatigue syndrome suggest selenium transport impairment and acquired resistance to thyroid hormone.

Redox biology (2023)

PMID: 37423160

DOI: 10.1016/j.redox.2023.102796

Cite This Figure

![Figure 6: Proposed pathogenic model linking anti-selenoprotein P autoantibodies to chronic fatigue syndrome symptoms through disrupted selenium transport, impaired deiodinase activity, and functional thyroid hormone resistance.]()

> Source: Qian Sun et al. "Autoantibodies to selenoprotein P in chronic fatigue syndrome suggest selenium t." *Redox biology*, 2023. PMID: [37423160](https://pubmed.ncbi.nlm.nih.gov/37423160/)

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  <img src="" alt="Proposed pathogenic model linking anti-selenoprotein P autoantibodies to chronic fatigue syndrome symptoms through disrupted selenium transport, impaired deiodinase activity, and functional thyroid hormone resistance." />
  <figcaption>Figure 6. Proposed pathogenic model linking anti-selenoprotein P autoantibodies to chronic fatigue syndrome symptoms through disrupted selenium transport, impaired deiodinase activity, and functional thyroid hormone resistance.<br>  Source: Qian Sun et al. "Autoantibodies to selenoprotein P in chronic fatigue syndrome suggest selenium t." <em>Redox biology</em>, 2023. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/37423160/">37423160</a></figcaption>
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