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Functional assay data demonstrating that anti-selenoprotein P autoantibodies inhibit selenium uptake in cell culture models. The blocking activity confirms a mechanistic link between autoantibodies and selenium transport impairment.

Figure 5

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Source Paper

Autoantibodies to selenoprotein P in chronic fatigue syndrome suggest selenium transport impairment and acquired resistance to thyroid hormone.

Redox biology (2023)

PMID: 37423160

DOI: 10.1016/j.redox.2023.102796

Cite This Figure

![Figure 5: Functional assay data demonstrating that anti-selenoprotein P autoantibodies inhibit selenium uptake in cell culture models. The blocking activity confirms a mechanistic link between autoantibodies and selenium transport impairment.]()

> Source: Qian Sun et al. "Autoantibodies to selenoprotein P in chronic fatigue syndrome suggest selenium t." *Redox biology*, 2023. PMID: [37423160](https://pubmed.ncbi.nlm.nih.gov/37423160/)

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  <img src="" alt="Functional assay data demonstrating that anti-selenoprotein P autoantibodies inhibit selenium uptake in cell culture models. The blocking activity confirms a mechanistic link between autoantibodies and selenium transport impairment." />
  <figcaption>Figure 5. Functional assay data demonstrating that anti-selenoprotein P autoantibodies inhibit selenium uptake in cell culture models. The blocking activity confirms a mechanistic link between autoantibodies and selenium transport impairment.<br>  Source: Qian Sun et al. "Autoantibodies to selenoprotein P in chronic fatigue syndrome suggest selenium t." <em>Redox biology</em>, 2023. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/37423160/">37423160</a></figcaption>
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